Pharmacology Assessment

20 learning units • 10 questions per unit • 200 explained questions

Module 06 — Adrenergic Agonists & Antagonists

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Question 01 • Single best answer

Which receptor most directly mediates salbutamol bronchodilation?

A. Alpha-1 B. Muscarinic M3 C. Nicotinic Nm D. Beta-2

Question 02 • Single best answer

Which action is most closely associated with alpha-1 stimulation?

A. Vascular smooth muscle contraction B. Reduced skeletal acetylcholine release C. Direct proton pump inhibition D. Platelet cyclooxygenase inhibition

Question 03 • Single best answer

Why may phenylephrine slow the heart despite raising blood pressure?

A. Baroreflex-mediated vagal activation B. Direct beta-1 blockade C. Acetylcholinesterase inhibition D. Destruction of the sinoatrial node

Question 04 • Single best answer

Which beta blocker is nonselective?

A. Metoprolol B. Bisoprolol C. Atenolol D. Propranolol

Question 05 • Single best answer

Why is nonselective beta blockade a concern in asthma?

A. It always increases surfactant secretion B. It directly dissolves mucus C. It blocks histamine H2 receptors D. It can oppose beta-2-mediated bronchodilation

Question 06 • Single best answer

Which medicine reduces sympathetic outflow through central alpha-2 activity?

A. Phenylephrine B. Dobutamine C. Salbutamol D. Clonidine

Question 07 • Single best answer

What may follow abrupt clonidine withdrawal?

A. Permanent paralysis B. Universal severe hypocalcemia C. Rebound hypertension D. Complete loss of adrenergic receptors

Question 08 • Single best answer

What is a characteristic early risk of prazosin therapy?

A. Severe cholinergic salivation B. Irreversible deafness in every patient C. Postural hypotension D. Direct respiratory muscle paralysis

Question 09 • Single best answer

What can beta blockers mask in hypoglycemia?

A. The glucose value measured in blood B. All sweating in every patient C. Adrenergic warning signs such as tachycardia D. The need for glucose metabolism

Question 10 • Single best answer

Why is alpha blockade established before beta blockade in specialist preparation for pheochromocytoma?

A. To activate platelet aggregation B. To increase intestinal drug absorption C. To abolish insulin secretion D. To avoid unopposed alpha-mediated vasoconstriction

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